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  • SFB 841
    • Description
    • Participating Institutions
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    • Liver.net Biobank
      • Sample collection
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  • Projects
    • A Projects
      • A2 Mechanisms underlying activation and polarisation of inflammatory monocytes and organ-specific regeneration in acute and chronic liver disease
      • A3 Regulation of antibacterial immune responses in the liver
      • A5 Molecular mechanisms of hepatitis B virus persistence in chronically infected human chimeric mice
      • A6 (Dys)-regulation of the cellular HCV-specific immune response
      • A7 HCV infection: Mechanisms of NK-cell-mediated control
      • A8 Hepatitis D Virus-induced pathogenesis and interaction with the immune system
    • B Projects
      • B1 Regulation of immune-mediated liver injury
      • B2 Induction and breaking of immune tolerance in the liver
      • B3 Regulatory T cells and TH17 cells in experimental cholangitis
      • B4 Regulation of the T cell response during malaria
      • B5 Phenotype and function of CD4+ T lymphocytes in autoimmune hepatitis
      • B6 The role of anti-inflammatory bile acids for the development and progression of non-alcoholic steatohepatitis (NASH)
      • B8 The contact system in liver: an interface of inflammation, innate immunity and coagulation
      • B9 Signalling pathways involved in the induction and function of myeloid-derived suppressor cells in liver inflammation
    • C Projects
      • C1 The role of gp130 signalling during liver regeneration, inflammation and tumour formation: therapeutic perspectives
      • C3 The role of IL-6 and NASH in hepatocellular carcinoma (HCC) linked to metabolic disease
      • C8 IL-22 and IL-22BP in liver regeneration and carcinogenesis
    • P Projects
      • P1 B-cells in autoimmune liver disease
      • P2 From apoptotic cell sensing to an innate memory response: a new aspect of Kupffer cell function
      • P3 Role of the aryl hydrocarbon receptor in hepatic immune regulation
      • P4 Progression from NAFLD to NASH: Miscommunication between the intestinal microbiota and CD4 T cells as a potential cause
    • SP Projects
      • SP1 MRI and MPI of liver disease - non-invasive control of liver and bile duct inflammation, tumourgenesis, steatosis, fibrosis and pathogenic parasites
      • SP2 CRISPR/Cas-mediated genome editing for the study of processes involved in liver inflammation and its consequences
      • SP3 Integrated Research Training Group "Inflammation & Regeneration"
    • T Project
      • Harnessing hepatic tolerance mechanisms for antigen-specific treatment of autoimmune and allergic diseases
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University of Hamburg
  • Center for Bioinformatics
  • Heinrich Pette Institute for Experimental Virology
University Medical Center Hamburg-Eppendorf
  • I. Department of Internal Medicine
  • Department of Experimental Immunology and Hepatology
  • Department for Cell and Gene Therapy, Clinic for Stem Cell Transplantation
  • Department of Biochemistry and Molecular Biology II: Molecular Cell Biology
  • Institute for Immunology
  • Department of Clinical Chemistry
  • Department of Cell Biochemistry and Clinical Neurobiology
  • Department of Pathology
  • Diagnostic and Interventional Radiology Department and Clinic

Bernhard Nocht Institute for Tropical Medicine

Christian-Albrechts-Universität zu Kiel
  • Institute of Biochemistry
Hadassah Medical Centre, Hebrew University of Jerusalem, Israel
  • Goldyne Savad Institute of Gene Therapy
ETH Zurich
  • Institute of Microbiology

DFG Deutsche Forschungsgemeinschaft

SFB841 - Sekretariat: Dr. Meike Zantz - I. Medizinische Klinik und Poliklinik - Universitätsklinikum Hamburg-Eppendorf - Martinistr. 52 - 20246 Hamburg Tel.: +49-40-7410-58653 - Fax: +49-40-7410-40156 - E-Mail: m.zantz@uke.de